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Delayed sleep–wake phase disorder
Also known as: Delayed sleep–wake phase disorder, Delayed sleep phase syndrome
Facts (31)
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Extent and Health Consequences of Chronic Sleep Loss and ... - NCBI ncbi.nlm.nih.gov 26 facts
referenceA 1988 study by Pelayo et al. published in Sleep Research estimated the prevalence of delayed sleep phase syndrome among adolescents.
claimDelayed sleep phase syndrome has a measurable prevalence among adolescents, as reported by Pelayo, Thorpy, and Govinsky in a 1988 study in Sleep Research.
claimIndividuals living in extreme latitudes who are exposed to extended periods of light may be at increased risk of suffering from delayed sleep phase syndrome.
referenceBright morning light acts as a treatment for delayed sleep phase syndrome by inducing phase-shifting effects, as reported in a 1990 study published in the journal Sleep.
claimWeyerbrock et al. (1996) investigated the effects of light and chronotherapy on human circadian rhythms, specifically examining cytokines, cortisol, growth hormone, and the sleep-wake cycle in patients with delayed sleep phase syndrome.
referenceB. Alvarez, M.J. Dahlitz, J. Vignau, and J.D. Parkes investigated clinical findings in 14 subjects with delayed sleep phase syndrome in a 1992 study published in the Journal of Neurology, Neurosurgery, and Psychiatry.
claimA study of 22 adolescents found that delayed sleep phase syndrome is associated with increased daytime irritability, poor school performance, and mental disturbances.
measurementIndividuals with delayed sleep phase syndrome typically have a normal total sleep time for their age, but find it difficult to initiate sleep before 2:00 a.m. to 6:00 a.m. and prefer to wake up between 10:00 a.m. and 1:00 p.m.
claimPolymorphisms in circadian genes influence the entraining and free-running period of the circadian cycle and may be associated with delayed sleep phase syndrome.
claimA study of 14 individuals reported that delayed sleep phase syndrome may impair job performance and may be associated with marital problems and financial difficulty.
referenceRegestein and Monk (1995) provided a clinical review of delayed sleep phase syndrome.
claimDelayed sleep phase syndrome is characterized by sleep onset and wake times that are typically delayed 3 to 6 hours relative to conventional sleep-wake times.
procedureTreatment for delayed sleep phase syndrome requires resynchronizing to a more appropriate phase to the 24-hour light-dark cycle, which may include a structured sleep-wake schedule, good sleep hygiene practices, and resetting the circadian pacemaker with bright light, melatonin, or a combination of both.
referenceTakahashi, Hohjoh, and Matsuura identified predisposing factors in delayed sleep phase syndrome, as published in Psychiatry and Clinical Neuroscience in 2000.
claimThere have been no large-scale controlled studies examining the efficacy of melatonin for treating delayed sleep phase syndrome, and it has not been approved by the Food and Drug Administration for this indication.
claimIndividuals with delayed sleep phase syndrome have a total sleep time that is normal for their age.
claimIt has been hypothesized that delayed sleep phase syndrome may result from a circadian phase with reduced sensitivity to photic entrainment or a prolonged free-running period of the circadian cycle.
claimA 1981 study by Weitzman, Czeisler, Coleman, Spielman, Zimmerman, Dement, Richardson, and Pollak identified delayed sleep phase syndrome as a chronobiological disorder characterized by sleep-onset insomnia.
claimDelayed sleep phase syndrome may be more prevalent in adolescents and young adults, although the exact prevalence is unclear.
claimNight shift workers may be at higher risk for delayed sleep phase syndrome due to irregular circadian entrainment.
claimIndividuals with delayed sleep phase syndrome exhibit a hypersensitivity to nighttime bright light exposure regarding the suppression of melatonin, despite the typical increase of melatonin levels in the evening hours.
claimStudies investigating the efficacy of bright light for treating delayed sleep phase syndrome have provided mixed results, partially due to limitations in study design and participant numbers, leading to a lack of standard criteria for its use.
referenceArcher SN, Robilliard DL, Skene DJ, Smits M, Williams A, Arendt J, and von Schantz M identified that a length polymorphism in the circadian clock gene Per3 is linked to delayed sleep phase syndrome and extreme diurnal preference, published in Sleep in 2003.
referenceA study on the estimated prevalence of delayed and advanced sleep phase syndromes was published in the journal Sleep Research in 1995.
claimA 2005 study published in the journal Sleep identified an association between the length polymorphism in the human Per3 gene and delayed sleep-phase syndrome.
referenceA 2003 study by Hohjoh et al. identified a significant association between the arylalkylamine N-acetyltransferase (AA-NAT) gene and delayed sleep phase syndrome.
Sleep Deprivation, Sleep Disorders, and Chronic Disease - CDC cdc.gov Aug 31, 2023 4 facts
claimPeople with Delayed sleep–wake phase disorder (DSWPD) often experience comorbid depression.
claimDelayed sleep–wake phase disorder (DSWPD) is often observed among adolescents and young adults and is characterized by a preference for sleep and wake times that are misaligned with societal demands.
claimDelayed sleep–wake phase disorder (DSWPD) is a condition characterized by a shift in the timing of sleep onset and offset, where sleep and wake times are substantially delayed relative to societal norms.
claimDelayed sleep–wake phase disorder (DSWPD) leads to distress or impairment in social, occupational, or other important areas of functioning.
Neuroscience of sleep - Wikipedia en.wikipedia.org 1 fact
referenceWeitzman et al. defined Delayed Sleep Phase Syndrome as a chronobiological disorder associated with sleep-onset insomnia in a 1981 study published in Archives of General Psychiatry.