In telomere-dysfunctional animals, administration of nicotinamide mononucleotide (NMN) preserves telomere length, inhibits the DNA damage response and p53, enhances mitochondrial activity, and reverses liver fibrosis.

Authors

• Person: Not available Organization: Nature
  Cellular rejuvenation: molecular mechanisms and potential ... - Nature

Sources

• Cellular rejuvenation: molecular mechanisms and potential ... - Nature www.nature.com Nature via serper

Referenced by nodes (2)

• telomere length concept
• DNA damage response concept